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Results Individuals carrying an APP pathogenic genetic variant harbor a metabolically dysfunctional subset of CD38-expressing CD4 + T cells prior to symptom onset In the present study, we focused on individuals of a newly described highly consanguineous family, belonging to a minority ethnic group in northern Israel

Biotinyl-glucose-6-phosphate dehydrogenase preparation, kinetics, and modulation by avidin

When interpreting these data, it needs to be considered that biochemical abnormalities may be consequences, rather that the cause, of prolonged inactivity

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2.2 HSC activation and liver fibrosis HSCs not only coordinate ECM deposition in the injured liver, but also are the main effectors of liver inflammation (Zhao and Chen, 2019)